Increasing fluid overload can impede the absorption of oral diuretics, compromising bioavailability.1-3 Through subcutaneous administration, FUROSCIX is able to deliver at-home, IV-equivalent diuresis with 99.6% absolute bioavailability—even in the presence of fluid overload.1* This allows you effective early intervention and your patients a return to maintenance diuretic therapy without ever leaving home.1
*
FUROSCIX is not for chronic use and should be replaced with oral diuretics as soon as practical.
In patients with reduced eGFR, furosemide plasma levels increased as renal function declined.
Mean Cmax values from FUROSCIX were lower than IV.4
Urine output between FUROSCIX and IV furosemide remained consistent regardless of eGFR.4
0-8 H
0-24 H
Furosemide can cause dehydration and azotemia. If increasing azotemia and oliguria occur during treatment of severe progressive renal disease, furosemide should be discontinued.
Open-label, crossover study design1:
†
90% confidence interval of 94.8-104.8. The design of the device used in this study was different from the current FUROSCIX On-Body Infusor, but it used the same administration profile.
‡
Based on mean ± SD 30-minute plasma concentration of 600 (± 209) ng/mL.1
§
Urine output over the periods of 0 to 8 hours and 0 to 24 hours following administration of 80-mg furosemide by 5-hour subcutaneous infusion with FUROSCIX, or IV administration of 2 doses of 40 mg 2 hours apart. Time 0 indicates the start of diuresis therapy.
Cmax=peak concentration; eGFR=estimated glomerular filtration rate; SD=standard deviation.